Wow this is a seriously serious supplementary figure from @cellpress.bsky.social today. Panels A through UU.
I get that Cell likes to minimize the number of supplementary figures. But is this really what is happening here?
www.cell.com/cell/fulltex...
Off to Dresden for my seminar tomorrow at @mpi-cbg.de kindly hosted by Sandra Scharaw. I can't wait to meet everyone and talk exciting science stuff 🔬🧪🧬. Presenting my Atlas of cytokinesis 😊 🌍🍬
John Prensner
Nadja M. Hümpfer
But what's the mechanism?
2nd postdoc job alert in the Crick Institute in London. Project will examine the role of motors & the cytoskeleton (actin, septins and microtubules) during vaccinia virus egress using biochemical, structural & cellular approaches + live cell imaging. spread the word.
crick.ac.uk/WayPostdoc20...
I tried an even harder example on Gemini Pro image generation and this is quite scary/amazing. I asked for a microscopy image of around 20 HeLa cells, GFP tagged 20% nuclear, 10% membrane, +1 nuclear staining, + overlap. Image below and prompt in the following post.
😂
#LivingArchitectures
We put cells and cytoskeleton filaments on the architecture of the musée d'Orsay.
www.musee-orsay.fr/fr/agenda/ev...
Scientists of the #CytoMorphoLab adapted their protocols to illustrate the questions that keep them awake at night.
-> Two shows on the 24th and 25th of January.
Today, our animation synthesizing decades of research on actin-mediated endocytosis in budding yeast was published:
journals.biologists.com/jcs/article/...
The result of a fantastic Iwasa-Drubin lab collaboration.
@margotriggi.bsky.social @jiwasa.bsky.social
movie.biologists.com/video/10.124...
MIT researchers have discovered how an immune system molecule triggers neurons in a specific brain circuit to shut down social behavior amid infection. picower.mit.edu/news/too-sic... @mitbcs.bsky.social #Neuroscience #behavior
Michael Way
Oded Rechavi
Pedro Beltrao
Prof Chris Jackson
Manuel Thery
David Drubin
MIT researchers have discovered how an immune system molecule triggers neurons in a specific brain circuit to shut down social behavior in mice modeling infection.
The Picower Institute for Learning and Memory at MIT
Now online! IL-1R1-positive dorsal raphe neurons drive self-imposed social withdrawal in sickness
During sickness, animals exhibit prolonged social withdrawal, a response that may limit disease spread. Elevated IL-1β levels during inflammation activate IL-1R1-expressing neurons in the dorsal raphe nucleus to drive self-imposed withdrawal, highlighting a neural mechanism through which immune activation reconfigures social behavior.
