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Please note that the zoom link for tomorrow's webinar with @nitikataneja.bsky.social and @doksanilab.bsky.social has been updated: uzh.zoom.us/j/6466274666...
Today we are very pleased to share our latest article available now online @narjournal.bsky.social about the role of Slx4 scaffold and Fun30 (SMARCAD1 homolog) in DNA damage response regulation and replication fork protection against Top1-DNA crosslinks. academic.oup.com/nar/article/...
Thank you to @doksanilab.bsky.social, @nitikataneja.bsky.social, and everyone who joined us for such a great discussion today! Weโ€™re excited to invite you to our next webinar on March 3rd with @centromellone.bsky.social and Michele Pagano.
Another great science appointment is coming up with @nitikataneja.bsky.social and @doksanilab.bsky.social! Join us live on Tuesday, February 3rd at 5pm (Rome) / 11am (NYC). Zoom link: uniroma1.zoom.us/j/95149984403 Register here to stay in the loop: docs.google.com/forms/d/e/1F...
4mo
2mo
4mo
4mo
Our lab is featured in the latest Erasmus MC Annual Report. Thanks to my team, our collaborators and funding agencies for their continued support & making this possible! ๐Ÿ™ Read here๐Ÿ‘‡: digitaalpubliceren.com/erasmusmc/21...
6d
Delighted to be part of this work from the Lopes lab identifying nucleoplasmic Lamin A/C as a key regulator of replication fork plasticity, acting via de novo heterochromatin and ADP-ribosylation to control RECQ1-mediated fork restart. Now in Nature Comm. ๐ŸŽ‰Special efforts: @joannapaulson.bsky.social
Among the anti-recombinases, FIGNL1 rules them all. So much that inactivating it brings BRCA2-deficient cells to life. Who is responsible for RAD51 loading without BRCA2/FIGNL1, check out the paper to find out! Great collaboration with @raychaudhurilab.bsky.social www.science.org/doi/10.1126/...
6mo
GIIN
GIIN
GIIN
7mo
Benjamin Pardo
Halloween borrel at the department ๐ŸŽƒ : our PhDs & Postdocs showed next-level creativity by revealing the hidden monstrous sides of their PIs! ๐Ÿ‘ป๐Ÿ˜‚ Absolutely hilarious & scary indeed๐Ÿ˜œโ€ฆ though Iโ€™m starting to wonder if thatโ€™s really how they see me ๐Ÿ‘€๐Ÿ˜…
Delighted & grateful to receive @erc.europa.eu support once again! This ERC-PoC will translate fundamental insights into therapy-induced chromatin reorganization toward proof-of-concept in ovarian cancer patient samples. Special thanks to @oncodeinstitute.bsky.social for continued support! ๐Ÿ™Œ
7mo
www.linkedin.com/posts/oncode...
Nature Communications - Replication fork plasticity upon genotoxic stress is modulated by nuclear architectural components by elusive mechanisms. Here the authors implicate Lamin A/C โ€“ best...
Nucleoplasmic Lamin A/C controls replication fork restart upon stress by modulating local H3K9me3 and ADP-ribosylation levels
rdcu.be
4mo
4mo
Nitika Taneja @TanejaLab
Nitika Taneja @TanejaLab
Nitika Taneja @TanejaLab
๐ŸšจGreat news: Oncode Investigators Nitika Taneja (Erasmus MC) and Sebastian Pomplun (LUMC) have been awarded European Research Council (ERC) Proof of Concept (PoC) funding. As European Commissioner E...
www.linkedin.com
#ercpoc #erc #oncodeinstitute #cancerresearch #ovariancancer | Oncode Institute
Petr Cejka
Nitika Taneja @TanejaLab
Nitika Taneja @TanejaLab
Thrilled to share our latest work - now available in full in Nature Communications - led in my lab by @vcherdy.bsky.social, with key collaborative contributions from @nitikataneja.bsky.social . rdcu.be/eVrxX
6mo
Nature Communications - Replication fork plasticity upon genotoxic stress is modulated by nuclear architectural components by elusive mechanisms. Here the authors implicate Lamin A/C โ€“ best...
rdcu.be
Nucleoplasmic Lamin A/C controls replication fork restart upon stress by modulating local H3K9me3 and ADP-ribosylation levels
Lopes_Lab