Passionate about studying #brainmetastases 🧠, #cancerneuroscience ⛓️💥, #microscopy🔬 and working with my #team 👏.
Team leader at the DKFZ and University Hospital Heidelberg, Germany
Matthia Karreman
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📢 #CancerNeuroscience Online Seminar with Manuel Valiente from CNIO, Madrid, Spain.
Title: Molecular and cellular rationale underlying the heterogeneous impact of metastases on brain activity
June 9, from 5-6 PM CEST, please register here: us06web.zoom.us/meeting/regi...
Cancer Neuroscience Heidelberg
Super excited to see our new preprint on calcium signalling in brain metastases now out on bioRxiv!
Very thankful to be part of @winklerlab.bsky.social and working so closely with @nilshebach.bsky.social under the guidance of @matthiakarreman.bsky.social and Frank Winkler and all our collaborators.
Happy #FluorescenceFriday!
Gap junction inhibition disconnects tumour networks in brain metastases and silences Ca²⁺ oscillations that drive proliferation.
More in our recent preprint : www.biorxiv.org/content/10.6...
Our new preprint on calcium signaling in brain metastases is now on bioRxiv!
Thrilled to share the core work of my doctoral thesis in the @winklerlab.bsky.social, together with @nielsolshausen.bsky.social, @matthiakarreman.bsky.social and many more!
Nils Hebach
Nils Hebach
Manuel Valiente, DVM, PhD - Molecular and cellular rationale underlying the heterogeneous impact of metastases on brain activity
What if brain metastatic cells do not act alone, but form #networks to help them grow?
In our new #preprint, we show that #brainmetastases form malignant, gap-junction mediated #networks that drive cell cycle progression & neural features through Ca²⁺ signaling.
www.biorxiv.org/content/10.6...
🧵👇
What if brain metastatic cells do not act alone, but form #networks to help them grow?
In our new #preprint, we show that #brainmetastases form malignant, gap-junction mediated #networks that drive cell cycle progression & neural features through Ca²⁺ signaling.
www.biorxiv.org/content/10.6...
🧵👇
