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This phenotype is driven by inflammatory cues (TGF-β/GM-CSF) and enhanced by G-CSF priming—highlighting tissue-driven immune heterogeneity. Way to go, Team. Check out the paper here: pubmed.ncbi.nlm.nih.gov/42019709/
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Myocardial infarction (MI) triggers an immune response marked by rapid infiltration of neutrophils. Although once considered a uniform population, neutrophils display remarkable heterogeneity post-MI,...
pubmed.ncbi.nlm.nih.gov
SiglecF-expressing neutrophils differentiate from mature neutrophils locally in the heart after myocardial infarction - PubMed
Center For Cardiometabolic Science