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If you’re interested in host–microbe interactions, immunometabolism, mucosal biology, or fungal pathogenesis, I hope you’ll check out the full article. 👇 @science.org www.science.org/doi/10.1126/...
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Candida hijacks epithelial metabolism via glycolysis and GOT1-dependent TCA shunt, influencing antifungal defense.
www.science.org
Fungal infection drives metabolic reprogramming in epithelial cells via aerobic glycolysis and an alternative TCA cycle shunt
Aize Pellón Rodríguez