Postdoc at University of Copenhagen trying to understand why some develop obesity while others are resistant to weight gain âïž
Jens Lund
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Grateful for the support @easoobesity.bsky.social - and the opportunity to present data on 1) âanti-obesityâ metabolites, 2) problems with the pyruvate tolerance test, and 3) feed efficiency as a driver of obesity from @clemmensenc.bsky.social and @zgerharthines.bsky.social labs!
@cbmr.science
Take the escalator down to the âmonkey businessâ in the bottom of ICC đ Resist the urge to enter the âdark sideâ with flashy advertisements for appetite-suppressing drugs đ 4/5
Hvis din omgangskreds stiller spĂžrgsmĂ„lstegn ved vigtigheden af grundforskning, sĂ„ vis dem dette gode klip med Adam Bencard đđŒ
Instead, go treasure-hunting in the enlightening poster area i.e the orange track 1 in the middle aisle đ here, you will find a postdoc, dressed like he should have been in Alanya, showcasing LBP1.031 đ„ 5/5
Metabolism Day strikes again đ„ Amazing line up of speakers - and fascinating data by Matthias Tschöp to kick off the day!
@cbmr.science
If you are at #ECO2026 in Istanbul, come see data indicating that differences in feed efficiency might be a main explanation for why inbred đ differ so much in HFD-induced weight gain âïž 2/5
Readworthy Letter in @cp-cellmetabolism.bsky.social on new-approach methods đ
"NAMs enhance and synergize with, rather than replace, studies in intact organisms. Animal research remains indispensable for most integrated, chronic, or behaviorally anchored questions."
www.cell.com/cell-metabol...
Over the last year, @federicopicciau.bsky.social in @clemmensenc.bsky.social Lab has been driving an exciting study in which we have meticulously monitored a big cohort of mice that develop various degrees of obesity - without overeating. 3/5
Online Now: A fuel partitioning perspective on appetite suppression by GLP-1 receptor agonists #trends #endocrinology #metabolism
Is diet-induced obesity driven by overeating? Or is there another and perhaps more important driver?đĄ1/5
@easoobesity.bsky.social @cbmr.science
Appetite suppression by glucagon-like peptide-1 receptor agonists (GLP-1RAs) is thought to result from direct actions on neural circuits controlling food intake. In this article, we (1) propose that a shift in fuel partitioning toward fat oxidation caused by GLP-1RAs contributes to appetite suppression and (2) explore potential lines of research to test this hypothesis.
